Growing evidence indicates that mitochondrial dysfunction is one of the key intracellular mechanisms associated with the pathogenesis of Alzheimer's disease. The role of mitochondria as regulators of energy metabolism and cell death pathways makes it crucial to neuronal cell survival or death. The goal of this conference was to critically examine potential drug therapeutics for mitochondrial dysfunction in Alzheimer’s disease. Recent findings from basic, clinical, and translational research were presented in a forum designed to stimulate discussion of mitochondrial function and its relationship to age-related neurodegenerative disease, in particular, Alzheimer’s disease and its treatment.
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